N-Acetyl Semax (NAS) is a synthetic heptapeptide derived from ACTH(4-10), investigated for its potential cognitive-enhancing, neuroprotective, and anxiolytic properties. It is believed to exert its ef...
N-Acetyl Semax (NAS) is a synthetic heptapeptide derived from ACTH(4-10), investigated for its potential cognitive-enhancing, neuroprotective, and anxiolytic properties. It is believed to exert its effects by modulating BDNF expression and potentially other neurotransmitter systems. It is primarily administered intranasally. While generally well-tolerated, common side effects may include headache and insomnia. Its use in stroke recovery is also being explored. More extensive research is required to fully elucidate its efficacy and safety profile across various applications.
Key Information
Inside Body Harmful Score
Common Dosage
Varies, often 200-600mcg daily, typically administered intranasally. Dosage ranges depend on individual response and intended use.
Source Type
synthetic
Production Process
Synthesized heptapeptide analog of ACTH(4-10)
Use Type
Inside BodyPotential Benefits
Cognitive enhancement improves mental performance by boosting memory, attention, and processing speed. It works by promoting neuronal health and protecting against cognitive decline.
Neuroprotection aims to protect the brain and nervous system from damage and degeneration by preventing neuronal cell death, reducing inflammation, and enhancing synaptic plasticity. Strategies include lifestyle modifications, nutritional supplementation, and pharmacological interventions to mitigate effects of aging, neurodegenerative diseases, stroke, and traumatic brain injury.
Alpha-GPC may improve stroke recovery by increasing acetylcholine levels, enhancing neuronal plasticity, and aiding in the repair of damaged neuronal structures. It potentially facilitates the brain's ability to reorganize and form new connections after a stroke.
L-tryptophan increases serotonin levels in the brain, potentially reducing anxiety and promoting relaxation. It influences mood regulation through its role as a serotonin precursor.
Citicoline protects brain cells by supporting neuronal membrane repair, enhancing cholinergic neurotransmission, and reducing oxidative stress and excitotoxicity. This helps to maintain and improve overall neuronal health.
Chrysin shows potential anxiolytic properties through interaction with benzodiazepine receptors, potentially reducing anxiety. Further research is needed to confirm these effects.
Possible Side Effects
Insomnia is a sleep disorder involving difficulty falling or staying asleep, leading to non-restorative sleep. It can be triggered by various factors and can result in daytime fatigue and impaired cognitive function.
Headache is pain in the head ranging from mild to severe, potentially with nausea, light/sound sensitivity, or dizziness. It can be caused by tension, dehydration, stress, or medication side effects, and persistent headaches require medical evaluation.
NALT may cause anxiety, restlessness, and insomnia, especially in sensitive individuals or those with pre-existing anxiety. These side effects are related to NALT's potential stimulant properties.
Scientific Evidence
Research papers and studies on PubMed and other scientific databases. Specific studies often focus on its effects on BDNF, cognitive function, and potential neuroprotective mechanisms. Keywords: N-Acetyl Semax, NAS, ACTH(4-10), BDNF, cognitive enhancement, neuroprotection, stroke.
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